2 edition of Maternal disorders related to fetal stress, perinatal death, and congenital defects found in the catalog.
Maternal disorders related to fetal stress, perinatal death, and congenital defects
National Institutes of Health (U.S.). Library Branch
Written in English
|Statement||Comp. by Elizabeth Koenig|
|Series||Public health bibliography series -- no. 25, Public Health Service publication -- no. 669|
|The Physical Object|
|Pagination||v, 33 p.|
|Number of Pages||33|
|LC Control Number||59062431|
Neonatal hypocalcemia owing to prematurity, perinatal stress/asphyxia, and maternal diabetes is common but is typically transient, Less common causes include maternal hyperparathyroidism, transient neonatal hypoparathyroidism, vitamin D deficiency, excessive diuretic use or phosphate load, and congenital hypoparathyroidism. Maternal vitamin D . Opioid use disorder among pregnant women is a significant public health concern in the United States. The number of pregnant women with opioid use disorder at labor and delivery more than quadrupled from to , according to a recent CDC use disorder during pregnancy has been linked with serious negative health outcomes for pregnant women and .
The rapid pace of fetal development by far exceeds any other stage of the life span, and thus, environmental influences can profoundly alter the developmental course. Stress during the prenatal period, including malnutrition and inflammation, impact maternal and fetal neurodevelopment with long-term consequences for physical and mental health of both the . Influences of maternal diabetes on fetal heart development: High blood sugar during pregnancy is associated with congenital heart defects. ScienceDaily. Retrieved August .
Maternal characteristics that were important predictors of fetal death 20 years ago, including diabetes, previous fetal death, and having a first child, are no longer independent risk factors. Hypertensive disorders and infection have been the leading causes of maternal death in the last 50 years in the United States. Maternal mortality is also more likely if the patient is less than 20 years of age. Migraine is not a life-threatening disease and does not affect pregnancy.
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Get this from a library. Maternal disorders related to fetal stress, perinatal death, and congenital defects: selected references, [Elizabeth Koenig; National Institutes of Health (U.S.).
Library Branch,]. CAUSES OF PERINATAL MORTALITY 27 compilation (Williams ). Toxemia and prematurity were more definite, the latter not more manageable however (recognized at that time to be due to “inability of the poorly developed child to lead an extra-uterine life,” and which now as then remains an important component of perinatal mortality).
Hypertension in pregnancy remains a leading cause of maternal death and perinatal and maternal morbidity worldwide. Improvements in management in recent years have Maternal disorders related to fetal stress reduced the risk of maternal death from hypertensive disorders in pregnancy, but a significant burden of morbidity and medically indicated premature delivery remains.
Maternal stress increases with receipt of a prenatal diagnosis of fetal congenital heart disease (CHD); however, the association between maternal stress and the developing brain in fetuses with CHD is unknown. Objective To determine the association of maternal psychological distress with brain development in fetuses with CHD.
Maternal Disorders related to Fetal Stress, Perinatal Death and Congenital Defects By Topics: Book Review. This report presents fetal and perinatal mortality data by maternal age, marital status, race, Hispanic origin, and state of residence, as well as by fetal birthweight, gestational age, plurality, and sex.
Trends in fetal and perinatal mortality are also examined. Methods— Descriptive tabulations of data are presented and interpreted. Maternal serum screening for neural tube defects, fetal ultrasound, and echocardiography should be performed Maternal disorders related to fetal stress the mid-trimester.
During gestation, the anticonvulsant levels should be checked monthly, and the dose should be adjusted accordingly, particularly with the use of lamotrigine, carbamazepine, and phenytoin (Harden et al., ).Author: Thomas F. Kelly. Maternal illnesses increase the chance that your baby will be born with a birth defect or have a chronic health problem.
Diabetes, cytomegalovirus, toxoplasmosis and Strep B are just a few of the illnesses that can cause an adverse outcome to pregnancy. Knowing you have these illnesses can go a long way to minimizing their effects. Congenital CMV And Birth Defects.
Cytomegalovirus, or CMV, is a very common member of the herpes family of viruses. By half of the adult population has been infected with CMV.
Significant anxiety has been demonstrated amongst women referred for fetal echocardiography.6, 7 Prenatal diagnosis of congenital malformations results in greater postnatal anxiety for women in comparison with those diagnosed solely after birth. 25 However, little research to date has focused on overall characterization of maternal stress.
After decades of decline, the maternal mortality rate in the United States has increased over the last 10 years. According to the Centers for Disease Control and Prevention (CDC), between and women in the United States die each year from pregnancy-related complications, illnesses or events.
Inthe U.S. maternal mortality rate (MMR)—the rate. Title(s): Maternal disorders related to fetal stress, perinatal death, and congenital defects; selected references, Comp. by Elizabeth Koenig. Country of Publication: United States Publisher: [Washington, ] Description: v, 33 p.
NLM ID: R[Book]. Seizure Disorder Effects on Pregnancy Increased risk of congenital anomalies (regardless if on or off meds) Cleft lip or palate Congenital heart defects Neural tube defects Children of Epileptic Patients Increased risk of neonatal death Decreased IQ Abnormal EEG patterns Early onset neonatal hemorrhagic disease (low Vit K).
Maternal death may occur due to the likelihood of severe hemorrhage (ante- and postpartum), shock, disseminated intravascular coagulation and renal failure. Perinatal mortality is mainly due to fetal hypoxia from the premature separation of the placenta, IUGR, low birth weight, congenital malformation, and fetal anemia.
Maternal diseases that cause specific fetal disease can do so by transplacental passage of a toxic maternal metabolic end product (e.g., high glucose or high androgen), by lack of transplacental passage of an essential maternal metabolic end product (e.g., thyroxine), or by transplacental passage of a maternal antibody.
Another study found a reduction in risk among women with symptoms of post-traumatic stress disorder (PTSD) or depression. The possible exception is a slight shortening of gestation reported in Dutch women post-9/11 who learned about the disaster through the media ( days, p=).
Studies of fetal growth have more varied results. MATERNAL DIABETES AND EVOLUTION OF CONGENITAL HEART DISEASE. While improvements in fetal surveillance and perinatal management have led to a reduction in diabetes related complications including perinatal mortality, the incidence of associated congenital anomalies remains high relative to the general population.
Therefore, it is expected that severe birth defects (such as complex congenital heart defects), placental insufficiency, maternal metabolic derangement (e.g. maternal ketoacidosis in DM1), and fetal hypoxia due to premature rupture of the membranes are leading causes of late pregnancy losses in DM.
However, literature data are scanty on this point. Background Pregnant women with congenital heart defects (CHDs) may be at increased risk for adverse events during delivery. Objectives This study sought to compare comorbidities and adverse cardiovascular, obstetric, and fetal events during delivery between pregnant women with and without CHDs in the United States.
Methods Comorbidities and adverse delivery events in. Introduction. Congenital and genetic disorders are a major cause of morbidity and premature death in childhood. The presentation of these conditions may be at or before birth with congenital malformations, in early life with impaired development, or in the older child with learning difficulties or problems with growth or sexual development.
Higher levels of flood-related maternal subjective stress, but not objective hardship, predicted worse theory of mind at 30 months (n =).
Further, maternal cognitive appraisal of the flood moderated the effects of stress in pregnancy on girls’ theory of mind performance but not boys’.VACTERL, the prototype for associated congenital anomalies, also has connections with functional issues such as pregnancy losses, prematurity, growth delays, perinatal difficulties, and parental subfertility.
This segues into a broader association with similar connections even in the absence of malformations. DNA methylation disturbances in the ovum are a likely cause, with .